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Retraction: Myostatin is a novel tumoral factor that induces cancer cachexia.

The Biochemical journal

[No authors listed]
PMID: 27060107
Biochem J. 2016 Apr 15;473(8):1111. doi: 10.1042/BJ4731111.

No abstract available.

Cite
Retraction: Myostatin is a novel tumoral factor that induces cancer cachexia. Biochem J. 2016;473(8):1111doi: 10.1042/BJ4731111.
(2016). Retraction: Myostatin is a novel tumoral factor that induces cancer cachexia. The Biochemical journal, 473(8), 1111. https://doi.org/10.1042/BJ4731111
"Retraction: Myostatin is a novel tumoral factor that induces cancer cachexia." The Biochemical journal vol. 473,8 (2016): 1111. doi: https://doi.org/10.1042/BJ4731111
Retraction: Myostatin is a novel tumoral factor that induces cancer cachexia. Biochem J. 2016 Apr 15;473(8):1111. doi: 10.1042/BJ4731111. PMID: 27060107; PMCID: PMC4948112.
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Identification of key binding site residues of MCT1 for AR-C155858 reveals the molecular basis of its isoform selectivity.

The Biochemical journal

Nancolas B, Sessions RB, Halestrap AP.
PMID: 25793420
Biochem J. 2015 Apr 01;467(1):192. doi: 10.1042/bj4670192.

No abstract available.

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Nancolas B, Sessions RB, Halestrap AP. Identification of key binding site residues of MCT1 for AR-C155858 reveals the molecular basis of its isoform selectivity. Biochem J. 2015;467(1):192doi: 10.1042/bj4670192.
Nancolas, B., Sessions, R. B., & Halestrap, A. P. (2015). Identification of key binding site residues of MCT1 for AR-C155858 reveals the molecular basis of its isoform selectivity. The Biochemical journal, 467(1), 192. https://doi.org/10.1042/bj4670192
Nancolas, B, et al. "Identification of key binding site residues of MCT1 for AR-C155858 reveals the molecular basis of its isoform selectivity." The Biochemical journal vol. 467,1 (2015): 192. doi: https://doi.org/10.1042/bj4670192
Nancolas B, Sessions RB, Halestrap AP. Identification of key binding site residues of MCT1 for AR-C155858 reveals the molecular basis of its isoform selectivity. Biochem J. 2015 Apr 01;467(1):192. doi: 10.1042/bj4670192. PMID: 25793420.
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Understanding the nature of health: New perspectives for medicine and public health. Improved wellbeing at lower costs.

F1000Research

Bircher J, Hahn EG.
PMID: 27134730
F1000Res. 2016 Feb 12;5:167. doi: 10.12688/f1000research.7849.1. eCollection 2016.

BACKGROUND: Current dilemmas of health care systems call for a new look at the nature of health. This is offered by the Meikirch model. We explore its hypothetical benefit for the future of medicine and public health. Meikirch model:...

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Bircher J, Hahn EG. Understanding the nature of health: New perspectives for medicine and public health. Improved wellbeing at lower costs. F1000Res. 2016;5:167doi: 10.12688/f1000research.7849.1.
Bircher, J., & Hahn, E. G. (2016). Understanding the nature of health: New perspectives for medicine and public health. Improved wellbeing at lower costs. F1000Research, 5167. https://doi.org/10.12688/f1000research.7849.1
Bircher, Johannes, and Hahn, Eckhart G. "Understanding the nature of health: New perspectives for medicine and public health. Improved wellbeing at lower costs." F1000Research vol. 5 (2016): 167. doi: https://doi.org/10.12688/f1000research.7849.1
Bircher J, Hahn EG. Understanding the nature of health: New perspectives for medicine and public health. Improved wellbeing at lower costs. F1000Res. 2016 Feb 12;5:167. doi: 10.12688/f1000research.7849.1. eCollection 2016. PMID: 27134730; PMCID: PMC4837984.
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Available Instruments for Analyzing Molecular Dynamics Trajectories.

The open biochemistry journal

Likhachev IV, Balabaev NK, Galzitskaya OV.
PMID: 27053964
Open Biochem J. 2016 Mar 14;10:1-11. doi: 10.2174/1874091X01610010001. eCollection 2016.

Molecular dynamics trajectories are the result of molecular dynamics simulations. Trajectories are sequential snapshots of simulated molecular system which represents atomic coordinates at specific time periods. Based on the definition, in a text format trajectory files are characterized by...

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Likhachev IV, Balabaev NK, Galzitskaya OV. Available Instruments for Analyzing Molecular Dynamics Trajectories. Open Biochem J. 2016;10:1-11doi: 10.2174/1874091X01610010001.
Likhachev, I. V., Balabaev, N. K., & Galzitskaya, O. V. (2016). Available Instruments for Analyzing Molecular Dynamics Trajectories. The open biochemistry journal, 101-11. https://doi.org/10.2174/1874091X01610010001
Likhachev, I V, et al. "Available Instruments for Analyzing Molecular Dynamics Trajectories." The open biochemistry journal vol. 10 (2016): 1-11. doi: https://doi.org/10.2174/1874091X01610010001
Likhachev IV, Balabaev NK, Galzitskaya OV. Available Instruments for Analyzing Molecular Dynamics Trajectories. Open Biochem J. 2016 Mar 14;10:1-11. doi: 10.2174/1874091X01610010001. eCollection 2016. PMID: 27053964; PMCID: PMC4797681.
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Minimally invasive surgical techniques in the management of differentiated thyroid cancer.

Otorinolaringologia

Tucker N, Mitchem J, Gillanders W.
PMID: 26504264
Otorinolaringologia. 2013 Jun;63(2):53-61.

Thyroid cancer is the fifth most common cancer in women with approximately 60,220 expected new cases in the United States in 2013, and the incidence continues to increase each year. Fortunately, the majority are well-differentiated thyroid cancers with excellent...

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Tucker N, Mitchem J, Gillanders W. Minimally invasive surgical techniques in the management of differentiated thyroid cancer. Otorinolaringologia. 2013;63(2):53-61
Tucker, N., Mitchem, J., & Gillanders, W. (2013). Minimally invasive surgical techniques in the management of differentiated thyroid cancer. Otorinolaringologia, 63(2), 53-61.
Tucker, N, et al. "Minimally invasive surgical techniques in the management of differentiated thyroid cancer." Otorinolaringologia vol. 63,2 (2013): 53-61.
Tucker N, Mitchem J, Gillanders W. Minimally invasive surgical techniques in the management of differentiated thyroid cancer. Otorinolaringologia. 2013 Jun;63(2):53-61. PMID: 26504264; PMCID: PMC4617759.
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Increased serum complement C3 and C4 concentrations and their relation to severity of chronic spontaneous urticaria and CRP concentration.

Journal of inflammation (London, England)

Kasperska-Zajac A, Grzanka A, Machura E, Misiolek M, Mazur B, Jochem J.
PMID: 23706062
J Inflamm (Lond). 2013 May 24;10(1):22. doi: 10.1186/1476-9255-10-22.

Chronic spontaneous urticaria (CU) is associated with activation of the acute phase response (APR). Nevertheless, APR-associated proteins have not been well characterized as potential biomarkers of the disease severity. To assess the pattern of complement proteins C3 and C4...

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Kasperska-Zajac A, Grzanka A, Machura E, et al. Increased serum complement C3 and C4 concentrations and their relation to severity of chronic spontaneous urticaria and CRP concentration. J Inflamm (Lond). 2013;10(1):22doi: 10.1186/1476-9255-10-22.
Kasperska-Zajac, A., Grzanka, A., Machura, E., Misiolek, M., Mazur, B., & Jochem, J. (2013). Increased serum complement C3 and C4 concentrations and their relation to severity of chronic spontaneous urticaria and CRP concentration. Journal of inflammation (London, England), 10(1), 22. https://doi.org/10.1186/1476-9255-10-22
Kasperska-Zajac, Alicja, et al. "Increased serum complement C3 and C4 concentrations and their relation to severity of chronic spontaneous urticaria and CRP concentration." Journal of inflammation (London, England) vol. 10,1 (2013): 22. doi: https://doi.org/10.1186/1476-9255-10-22
Kasperska-Zajac A, Grzanka A, Machura E, Misiolek M, Mazur B, Jochem J. Increased serum complement C3 and C4 concentrations and their relation to severity of chronic spontaneous urticaria and CRP concentration. J Inflamm (Lond). 2013 May 24;10(1):22. doi: 10.1186/1476-9255-10-22. PMID: 23706062; PMCID: PMC3680025.
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Phosphorylation of Ser158 regulates inflammatory redox-dependent hepatocyte nuclear factor-4a transcriptional activity.

The Biochemical journal

Guo H, Gao C, Mi Z, Wai PY, Kuo PC.
PMID: 24966053
Biochem J. 2014 Jul 15;461(2):347. doi: 10.1042/bj4610347.

No abstract available.

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Guo H, Gao C, Mi Z, et al. Phosphorylation of Ser158 regulates inflammatory redox-dependent hepatocyte nuclear factor-4a transcriptional activity. Biochem J. 2014;461(2):347doi: 10.1042/bj4610347.
Guo, H., Gao, C., Mi, Z., Wai, P. Y., & Kuo, P. C. (2014). Phosphorylation of Ser158 regulates inflammatory redox-dependent hepatocyte nuclear factor-4a transcriptional activity. The Biochemical journal, 461(2), 347. https://doi.org/10.1042/bj4610347
Guo, H, et al. "Phosphorylation of Ser158 regulates inflammatory redox-dependent hepatocyte nuclear factor-4a transcriptional activity." The Biochemical journal vol. 461,2 (2014): 347. doi: https://doi.org/10.1042/bj4610347
Guo H, Gao C, Mi Z, Wai PY, Kuo PC. Phosphorylation of Ser158 regulates inflammatory redox-dependent hepatocyte nuclear factor-4a transcriptional activity. Biochem J. 2014 Jul 15;461(2):347. doi: 10.1042/bj4610347. PMID: 24966053; PMCID: PMC4847221.
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Anticipatory role of high density lipoprotein and endothelial dysfunction: an overview.

The open biochemistry journal

Eren E, Yılmaz N, Aydin O, Ellidağ HY.
PMID: 25598849
Open Biochem J. 2014 Dec 31;8:100-6. doi: 10.2174/1874091X01408010100. eCollection 2014.

High Density Lipoprotein (HDL) has been witnessed to possess a range of different functions that contribute to its atheroprotective effects. These functions are: the promotion of macrophage cholesterol efflux, reverse cholesterol transport, anti-inflammatory, anti-thrombotic, anti-apoptotic, pro-fibrinolytic and anti-oxidative functions....

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Eren E, Yılmaz N, Aydin O, et al. Anticipatory role of high density lipoprotein and endothelial dysfunction: an overview. Open Biochem J. 2014;8:100-6doi: 10.2174/1874091X01408010100.
Eren, E., Yılmaz, N., Aydin, O., & Ellidağ, H. Y. (2014). Anticipatory role of high density lipoprotein and endothelial dysfunction: an overview. The open biochemistry journal, 8100-6. https://doi.org/10.2174/1874091X01408010100
Eren, Esin, et al. "Anticipatory role of high density lipoprotein and endothelial dysfunction: an overview." The open biochemistry journal vol. 8 (2014): 100-6. doi: https://doi.org/10.2174/1874091X01408010100
Eren E, Yılmaz N, Aydin O, Ellidağ HY. Anticipatory role of high density lipoprotein and endothelial dysfunction: an overview. Open Biochem J. 2014 Dec 31;8:100-6. doi: 10.2174/1874091X01408010100. eCollection 2014. PMID: 25598849; PMCID: PMC4293742.
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Photosystem II Repair and Plant Immunity: Lessons Learned from Arabidopsis Mutant Lacking the THYLAKOID LUMEN PROTEIN 18.3.

Frontiers in plant science

Järvi S, Isojärvi J, Kangasjärvi S, Salojärvi J, Mamedov F, Suorsa M, Aro EM.
PMID: 27064270
Front Plant Sci. 2016 Mar 31;7:405. doi: 10.3389/fpls.2016.00405. eCollection 2016.

Chloroplasts play an important role in the cellular sensing of abiotic and biotic stress. Signals originating from photosynthetic light reactions, in the form of redox and pH changes, accumulation of reactive oxygen and electrophile species or stromal metabolites are...

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Järvi S, Isojärvi J, Kangasjärvi S, et al. Photosystem II Repair and Plant Immunity: Lessons Learned from Arabidopsis Mutant Lacking the THYLAKOID LUMEN PROTEIN 18.3. Front Plant Sci. 2016;7:405doi: 10.3389/fpls.2016.00405.
Järvi, S., Isojärvi, J., Kangasjärvi, S., Salojärvi, J., Mamedov, F., Suorsa, M., & Aro, E. M. (2016). Photosystem II Repair and Plant Immunity: Lessons Learned from Arabidopsis Mutant Lacking the THYLAKOID LUMEN PROTEIN 18.3. Frontiers in plant science, 7405. https://doi.org/10.3389/fpls.2016.00405
Järvi, Sari, et al. "Photosystem II Repair and Plant Immunity: Lessons Learned from Arabidopsis Mutant Lacking the THYLAKOID LUMEN PROTEIN 18.3." Frontiers in plant science vol. 7 (2016): 405. doi: https://doi.org/10.3389/fpls.2016.00405
Järvi S, Isojärvi J, Kangasjärvi S, Salojärvi J, Mamedov F, Suorsa M, Aro EM. Photosystem II Repair and Plant Immunity: Lessons Learned from Arabidopsis Mutant Lacking the THYLAKOID LUMEN PROTEIN 18.3. Front Plant Sci. 2016 Mar 31;7:405. doi: 10.3389/fpls.2016.00405. eCollection 2016. PMID: 27064270; PMCID: PMC4814454.
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OBITUARY NOTICE: DAVID KEILIN (1887-1963).

The Biochemical journal

HARTREE EF.
PMID: 14097361
Biochem J. 1963 Oct;89:1-5. doi: 10.1042/bj0890001.

No abstract available.

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HARTREE EF. OBITUARY NOTICE: DAVID KEILIN (1887-1963). Biochem J. 1963;89:1-5doi: 10.1042/bj0890001.
HARTREE, E. F. (1963). OBITUARY NOTICE: DAVID KEILIN (1887-1963). The Biochemical journal, 891-5. https://doi.org/10.1042/bj0890001
HARTREE, E F. "OBITUARY NOTICE: DAVID KEILIN (1887-1963)." The Biochemical journal vol. 89 (1963): 1-5. doi: https://doi.org/10.1042/bj0890001
HARTREE EF. OBITUARY NOTICE: DAVID KEILIN (1887-1963). Biochem J. 1963 Oct;89:1-5. doi: 10.1042/bj0890001. PMID: 14097361; PMCID: PMC1202263.
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Long remissions in Hodgkin's disease.

Acta radiologica

BICHEL J.
PMID: 13275330
Acta radiol. 1955 Oct;44(4):325-36. doi: 10.3109/00016925509171293.

No abstract available.

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BICHEL J. Long remissions in Hodgkin's disease. Acta radiol. 1955;44(4):325-36doi: 10.3109/00016925509171293.
BICHEL, J. (1955). Long remissions in Hodgkin's disease. Acta radiologica, 44(4), 325-36. https://doi.org/10.3109/00016925509171293
BICHEL, J. "Long remissions in Hodgkin's disease." Acta radiologica vol. 44,4 (1955): 325-36. doi: https://doi.org/10.3109/00016925509171293
BICHEL J. Long remissions in Hodgkin's disease. Acta radiol. 1955 Oct;44(4):325-36. doi: 10.3109/00016925509171293. PMID: 13275330.
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An experiment in teaching family dynamics.

The American journal of psychiatry

FLINT AA, RIOCH MJ.
PMID: 13945554
Am J Psychiatry. 1963 Apr;119:940-4. doi: 10.1176/ajp.119.10.940.

No abstract available.

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FLINT AA, RIOCH MJ. An experiment in teaching family dynamics. Am J Psychiatry. 1963;119:940-4doi: 10.1176/ajp.119.10.940.
FLINT, A. A., & RIOCH, M. J. (1963). An experiment in teaching family dynamics. The American journal of psychiatry, 119940-4. https://doi.org/10.1176/ajp.119.10.940
FLINT, A A, and RIOCH, M J. "An experiment in teaching family dynamics." The American journal of psychiatry vol. 119 (1963): 940-4. doi: https://doi.org/10.1176/ajp.119.10.940
FLINT AA, RIOCH MJ. An experiment in teaching family dynamics. Am J Psychiatry. 1963 Apr;119:940-4. doi: 10.1176/ajp.119.10.940. PMID: 13945554.
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Showing 1 to 12 of 7268 entries